M. Pabst

Active 1973–2003

21
Papers
21,987
Citations
21
h-index
21
i10-index

Citations

Citations per year for M. Pabst1974: 8 citations1975: 26 citations1976: 17 citations1977: 20 citations1978: 36 citations1979: 27 citations1980: 23 citations1981: 24 citations1982: 17 citations1983: 24 citations1984: 43 citations1985: 37 citations1986: 36 citations1987: 33 citations1988: 47 citations1989: 34 citations1990: 49 citations1991: 54 citations1992: 47 citations1993: 57 citations1994: 46 citations1995: 42 citations1996: 38 citations1997: 53 citations1998: 56 citations1999: 74 citations2000: 76 citations2001: 105 citations2002: 91 citations2003: 113 citations2004: 89 citations2005: 109 citations2006: 99 citations2007: 100 citations2008: 103 citations2009: 109 citations2010: 92 citations2011: 106 citations2012: 95 citations2013: 65 citations2014: 70 citations2015: 57 citations2016: 49 citations2017: 51 citations2018: 40 citations2019: 332 citations2020: 326 citations2021: 270 citations2022: 156 citations2023: 94 citations2024: 225 citations2025: 72 citations

Citation sources

Countries

World map of the countries and regions citing this authorUnited States: 878 citing papers, 17.3% of this breakdownIndia: 740 citing papers, 14.6% of this breakdownEgypt: 325 citing papers, 6.4% of this breakdownBrazil: 245 citing papers, 4.8% of this breakdownChina: 231 citing papers, 4.6% of this breakdownUnited Kingdom: 213 citing papers, 4.2% of this breakdownNigeria: 192 citing papers, 3.8% of this breakdownGermany: 176 citing papers, 3.5% of this breakdownSaudi Arabia: 151 citing papers, 3% of this breakdownFrance: 133 citing papers, 2.6% of this breakdownJapan: 133 citing papers, 2.6% of this breakdownTürkiye: 126 citing papers, 2.5% of this breakdown
0%17.3%Other 30.1%

Fields

  • Biochemistry, Genetics and Molecular Biology27.9%
  • Medicine26.4%
  • Agricultural and Biological Sciences12.3%
  • Environmental Science11.5%
  • Immunology and Microbiology6.6%
  • Pharmacology, Toxicology and Pharmaceutics6%
  • Other9.3%

Topics

  • Glutathione Transferases and Polymorphisms5.3%
  • Genomics, phytochemicals, and oxidative stress5.1%
  • Environmental Toxicology and Ecotoxicology2.9%
  • Drug-Induced Hepatotoxicity and Protection2.2%
  • Selenium in Biological Systems1.6%
  • Aquaculture disease management and microbiota1.5%
  • Other81.4%

Coauthors

All papers

Open in search
  1. Glutathione S-Transferases

    Authors: , , - Journal of Biological Chemistry 1974 cited by 18,064

  2. Removal of endotoxin from protein solutions by phase separation using triton X-114

    Authors: , - Journal of Immunological Methods 1990 cited by 607

  3. The Identity of Glutathione S -Transferase B with Ligandin, a Major Binding Protein of Liver

    Authors: , , , , , - National Academy of Sciences, Proceedings of the National Academy of Sciences 1974 cited by 527

  4. Glutathione S-Transferase A

    Authors: , , - Journal of Biological Chemistry 1974 cited by 348

  5. Glutathione S-transferase AA from rat liver

    Authors: , , - Archives of Biochemistry and Biophysics 1976 cited by 215

  6. Inhibition of Neutrophil and Monocyte Defensive Functions by Nicotine

    Authors: , , , , , , , - Journal of Periodontology 1995 cited by 198

  7. Invasive M1T1 group A Streptococcus undergoes a phase‐shift in vivo to prevent proteolytic degradation of multiple virulence factors by SpeB

    Authors: , , , , , , - Molecular Microbiology 2003 cited by 194

  8. Activation of macrophages for enhanced release of superoxide anion and greater killing of Candida albicans by injection of muramyl dipeptide.

    Authors: , , - The Journal of Experimental Medicine 1980 cited by 117

  9. Enhanced release of oxygen metabolites by monocyte-derived macrophages exposed to proteolytic enzymes: activity of neutrophil elastase and cathepsin G.

    Authors: , , , , - The Journal of Immunology 1984 cited by 75

  10. Increased production of superoxide anion by macrophages exposed in vitro to muramyl dipeptide or lipopolysaccharide.

    Authors: , - The Journal of Experimental Medicine 1980 cited by 355

  11. Lipopolysaccharide priming of human neutrophils for an enhanced respiratory burst. Role of intracellular free calcium.

    Authors: , , , - Journal of Clinical Investigation 1989 cited by 213

  12. Cultured human monocytes require exposure to bacterial products to maintain an optimal oxygen radical response.

    Authors: , , - The Journal of Immunology 1982 cited by 144

  13. Chronic infection due to Mycobacterium intracellulare in mice: association with macrophage release of prostaglandin E2 and reversal by injection of indomethacin, muramyl dipeptide, or interferon-gamma.

    Authors: , , , , , - The Journal of Immunology 1986 cited by 143

  14. Inhibition of macrophage priming by sulfatide from Mycobacterium tuberculosis .

    Authors: , , , - The Journal of Immunology 1988 cited by 136

  15. Activation of mouse peritoneal macrophages by lipopolysaccharide alters the kinetic parameters of the superoxide-producing NADPH oxidase.

    Authors: , , - Journal of Biological Chemistry 1983 cited by 119

  16. Priming of neutrophils and macrophages for enhanced release of superoxide anion by the calcium ionophore ionomycin. Implications for regulation of the respiratory burst.

    Authors: , , , , , , - Journal of Biological Chemistry 1987 cited by 107

  17. Mercapturic acid formation: The several glutathione transferases of rat liver

    Authors: , , - Biochemical and Biophysical Research Communications 1973 cited by 98

  18. Priming of neutrophils by lipopolysaccharide for enhanced release of superoxide. Requirement for plasma but not for tumor necrosis factor-alpha.

    Authors: , - The Journal of Immunology 1990 cited by 89

  19. IFN-gamma and LPS overcome glucocorticoid inhibition of priming for superoxide release in human monocytes. Evidence that secretion of IL-1 and tumor necrosis factor-alpha is not essential for monocyte priming.

    Authors: , , , , , - The Journal of Immunology 1989 cited by 81

  20. CD18-deficient cells respond to lipopolysaccharide in vitro.

    Authors: , , , , , , , - The Journal of Immunology 1990 cited by 79

  21. Monocyte responses to sulfatide from Mycobacterium tuberculosis: inhibition of priming for enhanced release of superoxide, associated with increased secretion of interleukin-1 and tumor necrosis factor alpha, and altered protein phosphorylation

    Authors: , , , , - Infection and Immunity 1991 cited by 78

All 21 papers shown.