Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation of fibroblast-like synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 pathways

Yi Qin; Ming-Long Cai; Hui-Zhi Jin; Wei Huang; Zhu Chen; Aline Bözec; Jingang Huang; Zhu Chen

doi:10.1136/ard-2022-222605

Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation of fibroblast-like synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 pathways

Authors: Yi Qin, Ming-Long Cai, Hui-Zhi Jin, Wei Huang, Zhu Chen, Aline Bözec, Jingang Huang, Zhu Chen - Annals of the Rheumatic Diseases 2022 cited by 158

Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation of fibroblast-like synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 pathways | Litlas