Seigo Izumo

Active 1986–2010

102
Papers
30,397
Citations
91
h-index
102
i10-index

Citations

Citations per year for Seigo Izumo1955: 5 citations1986: 3 citations1987: 24 citations1988: 33 citations1989: 53 citations1990: 106 citations1991: 94 citations1992: 78 citations1993: 93 citations1994: 139 citations1995: 262 citations1996: 245 citations1997: 315 citations1998: 359 citations1999: 373 citations2000: 435 citations2001: 406 citations2002: 412 citations2003: 470 citations2004: 407 citations2005: 343 citations2006: 392 citations2007: 350 citations2008: 326 citations2009: 291 citations2010: 315 citations2011: 328 citations2012: 288 citations2013: 201 citations2014: 256 citations2015: 205 citations2016: 207 citations2017: 182 citations2018: 153 citations2019: 448 citations2020: 432 citations2021: 382 citations2022: 273 citations2023: 198 citations2024: 302 citations2025: 107 citations2026: 1 citations1956–1985: no citations, so these years are not shown

Citation sources

Countries

World map of the countries and regions citing this authorUnited States: 4,045 citing papers, 40.6% of this breakdownChina: 785 citing papers, 7.9% of this breakdownJapan: 617 citing papers, 6.2% of this breakdownGermany: 574 citing papers, 5.8% of this breakdownUnited Kingdom: 510 citing papers, 5.1% of this breakdownCanada: 453 citing papers, 4.6% of this breakdownItaly: 343 citing papers, 3.4% of this breakdownNetherlands: 290 citing papers, 2.9% of this breakdownFrance: 272 citing papers, 2.7% of this breakdownAustralia: 267 citing papers, 2.7% of this breakdownSpain: 183 citing papers, 1.8% of this breakdownSwitzerland: 163 citing papers, 1.6% of this breakdown
0%40.6%Other 14.7%

Fields

  • Medicine47.4%
  • Biochemistry, Genetics and Molecular Biology46.2%
  • Neuroscience2.1%
  • Immunology and Microbiology1.7%
  • Engineering0.8%
  • Agricultural and Biological Sciences0.3%
  • Other1.5%

Topics

  • Cardiac Fibrosis and Remodeling5.2%
  • Congenital heart defects research5%
  • Cardiomyopathy and Myosin Studies3.1%
  • Cardiovascular Function and Risk Factors2.4%
  • Cardiac electrophysiology and arrhythmias2.3%
  • Signaling Pathways in Disease2.1%
  • Other79.9%

Coauthors

All papers

Open in search
  1. Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

    Authors: , , , , , , , , , , , , , - Nature Medicine 2007 cited by 2,191

  2. Inhibition of mTOR Signaling With Rapamycin Regresses Established Cardiac Hypertrophy Induced by Pressure Overload

    Authors: , , , , , , - Circulation 2004 cited by 522

  3. Molecular characterization of angiotensin II-induced hypertrophy of cardiac myocytes and hyperplasia of cardiac fibroblasts. Critical role of the AT1 receptor subtype.

    Authors: , - Circulation Research 1993 cited by 1,421

  4. Reduced Phosphoinositide 3-Kinase (p110α) Activation Increases the Susceptibility to Atrial Fibrillation

    Authors: , , , , , , , , , , , , , , , , , - American Journal Of Pathology 2009 cited by 187

  5. Rapamycin Attenuates Load-Induced Cardiac Hypertrophy in Mice

    Authors: , , , , , , - Circulation 2003 cited by 463

  6. Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitro

    Authors: , , , - Cell 1993 cited by 1,354

  7. Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy

    Authors: , , , , , , - National Academy of Sciences, Proceedings of the National Academy of Sciences 2003 cited by 530

  8. Mechanism of endothelial cell shape change and cytoskeletal remodeling in response to fluid shear stress

    Authors: , - Journal of Cell Science 1996 cited by 525

  9. The conserved phosphoinositide 3‐kinase pathway determines heart size in mice

    Authors: , , , , , , , - The EMBO Journal 2000 cited by 606

  10. The cardiac homeobox gene Csx/Nkx2.5 lies genetically upstream of multiple genes essential for heart development

    Authors: , , , , - Development 1999 cited by 563

  11. Mouse cardiac surgery: comprehensive techniques for the generation of mouse models of human diseases and their application for genomic studies

    Authors: , , , , , - Physiological Genomics 2004 cited by 318

  12. Protective effects of exercise and phosphoinositide 3-kinase(p110α) signaling in dilated and hypertrophic cardiomyopathy

    Authors: , , , , , , , , , , , , , - National Academy of Sciences, Proceedings of the National Academy of Sciences 2007 cited by 295

  13. Morphogenesis of the right ventricle requires myocardial expression of Gata4

    Authors: , , , , , , - Journal of Clinical Investigation 2005 cited by 270

  14. Beneficial Effects of Mammalian Target of Rapamycin Inhibition on Left Ventricular Remodeling After Myocardial Infarction

    Authors: , , , , , , , , , , , - Journal of the American College of Cardiology 2009 cited by 213

  15. Regulation of Myocardial Contractility and Cell Size by Distinct PI3K-PTEN Signaling Pathways

    Authors: , , , , , , , , , , , , , , , , , , , , , , - Cell 2002 cited by 580

  16. The Insulin-like Growth Factor 1 Receptor Induces Physiological Heart Growth via the Phosphoinositide 3-Kinase(p110α) Pathway

    Authors: , , , , , , , , , , , , - Journal of Biological Chemistry 2004 cited by 400

  17. A Molecular Pathway Including Id2, Tbx5, and Nkx2-5 Required for Cardiac Conduction System Development

    Authors: , , , , , , , , , , , - Cell 2007 cited by 291

  18. Lipopolysaccharide-induced myocardial protection against ischaemia/reperfusion injury is mediated through a PI3K/Akt-dependent mechanism

    Authors: , , , , , , , , , , , , - Cardiovascular Research 2008 cited by 167

  19. PI3K(p110α) Protects Against Myocardial Infarction-Induced Heart Failure

    Authors: , , , , , , , , , , , , , , , , , - Arteriosclerosis Thrombosis and Vascular Biology 2010 cited by 171

  20. A Conserved Role for Phosphatidylinositol 3-Kinase but Not Akt Signaling in Mitochondrial Adaptations that Accompany Physiological Cardiac Hypertrophy

    Authors: , , , , , , , , , , , , , , , , - Cell Metabolism 2007 cited by 137

  21. FOG-2, a Cofactor for GATA Transcription Factors, Is Essential for Heart Morphogenesis and Development of Coronary Vessels from Epicardium

    Authors: , , , , , , , - Cell 2000 cited by 413

  22. Transgenic expression of green fluorescence protein can cause dilated cardiomyopathy

    Authors: , , , - Nature Medicine 2000 cited by 280

  23. Gata4 is required for maintenance of postnatal cardiac function and protection from pressure overload-induced heart failure

    Authors: , , , , , , , , , , , , - National Academy of Sciences, Proceedings of the National Academy of Sciences 2006 cited by 186

  24. Apoptosis in heart: basic mechanisms and implications in cardiovascular diseases

    Authors: , - Trends in Molecular Medicine 2003 cited by 151